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Talzenna

talazoparib
PARP Inhibitor Pfizer FDA Approved 2018
Indications Dosing Forms Contraindications Warnings Adverse Reactions Pharmacology Clinical Studies Tumor Types
1. Indications and Usage

Deleterious or suspected deleterious germline BRCA-mutated (gBRCAm) HER2-negative locally advanced or metastatic breast cancer, as detected by an FDA-approved test; Metastatic castration-resistant prostate cancer (mCRPC) with HRR gene mutations, in combination with enzalutamide.

2. Dosage and Administration

Breast cancer: 1 mg orally once daily
Prostate cancer: 0.5 mg orally once daily (with enzalutamide 160 mg daily)
Take with or without food. Swallow capsules whole.
Dose reductions (breast): 0.75 mg → 0.5 mg → 0.25 mg
Renal (CrCl 30-59): 0.75 mg daily (breast), 0.35 mg daily (prostate)

3. Dosage Forms and Strengths

Capsules: 0.25 mg, 1 mg

4. Contraindications

None listed.

5. Warnings and Precautions
  • Myelosuppression: Grade 3-4 anemia (39%), neutropenia (21%), thrombocytopenia (15%). Monitor CBC monthly. Dose reduce, hold, or D/C per severity.
  • Myelodysplastic Syndrome/Acute Myeloid Leukemia: MDS/AML in 0.5%. Monitor CBC.
  • Embryo-Fetal Toxicity: Can cause fetal harm. Effective contraception required during and 7 months after.
6. Adverse Reactions
Most Common Adverse Reactions

Fatigue (62%), anemia (53%), nausea (49%), neutropenia (35%), headache (33%), thrombocytopenia (27%), vomiting (25%), alopecia (25%), diarrhea (22%), decreased appetite (21%), abdominal pain (19%)

Fatigue
62%
Anemia
53%
Nausea
49%
Neutropenia
35%
Headache
33%
Thrombocytopenia
27%
Vomiting
25%
Alopecia
25%
Diarrhea
22%
Decreased Appetite
21%

Consult the complete prescribing information for a comprehensive list of adverse reactions and their frequencies.

12. Clinical Pharmacology
Mechanism of Action

Talazoparib inhibits poly(ADP-ribose) polymerase (PARP) enzymes, including PARP1 and PARP2, which are involved in DNA damage repair. Talazoparib is the most potent PARP trapper among approved PARP inhibitors — it not only inhibits PARP enzymatic activity (catalytic inhibition) but also stabilizes PARP-DNA complexes (PARP trapping), preventing DNA repair, replication fork progression, and causing cell death. Cancer cells with BRCA1/2 mutations or other HRR deficiencies are especially dependent on PARP for DNA repair, creating synthetic lethality when PARP is inhibited.

Pharmacokinetics

Tmax: 1-2 hours. Half-life: approximately 90 hours (long). Protein binding: 74%. Minimally metabolized (no significant CYP involvement). Excreted in urine (69%, 55% unchanged) and feces (20%). P-gp substrate. Steady-state by Week 3.

14. Clinical Studies

Clinical efficacy and safety data supporting the approval are available in the full prescribing information and from the clinical trials listed below.

Pivotal Clinical Trials
Additional Resources
Approved Tumor Types